Potential Influence of Exercise-Induced Rhabdomyolysis by Branched-Chain Amino Acid Supplementation
Abstract
No Abstract
1. da Luz CR, Nicastro H, Zanchi NE et al (2011). Potential therapeutic effects of branched-chain amino acids supplemen-tation on resistance exercise-based mus-cle damage in humans. J Int Soc Sports Nutr, 8: 23.
2. Sharp CP, Pearson DR (2010). Amino acid supplements and recovery from high-intensity resistance training. J Strength Cond Res, 24(4): 1125-1130.
3. Zheng L, Wei H, He P et al (2016). Effects of Supplementation of Branched-Chain Amino Acids to Reduced-Protein Diet on Skeletal Muscle Protein Synthesis and Degradation in the Fed and Fasted States in a Piglet Model. Nutrients, 9(1): E17.
4. Contrusciere V, Paradisi S, Matteucci A et al (2010). Branched-chain amino acids in-duce neurotoxicity in rat cortical cultures. Neurotox Res, 17(4): 392-398.
5. Fernstrom JD (2005). Branched-chain ami-no acids and brain function. J Nutr, 135(6 Suppl): 1539S-1546S.
6. Kim J, Lee J, Kim S et al (2016). Exercise-induced rhabdomyolysis mechanisms and prevention: A literature review. J Sport Health Sci, 5(3): 324-333.
7. Sung DJ, Lee M, Park JK et al (2018). Com-bination of Antidepressant and Alcohol Intake as a Potential Risk Factor for Rhabdomyolysis. Iran J Public Health, 47(9): 1424-1425.
8. Holt, SG, Moore KP (2001). Pathogenesis and treatment of renal dysfunction in rhabdomyolysis. Intensive Care Med, 27(5): 803-811.
9. Zhenyukh O, Civantos E, Ruiz-Ortega M et al (2017). High concentration of branched-chain amino acids promotes oxidative stress, inflammation and migra-tion of human peripheral blood mono-nuclear cells via mTORC1 activation. Free Radical Biol Med, 104: 165-177.
10. Li T, Zhang Z, Kolwicz SC Jr et al (2017). Defective Branched-Chain Amino Acid Catabolism Disrupts Glucose Metabo-lism and Sensitizes the Heart to Ischemia-Reperfusion Injury. Cell Metab, 25(2): 374-385.
2. Sharp CP, Pearson DR (2010). Amino acid supplements and recovery from high-intensity resistance training. J Strength Cond Res, 24(4): 1125-1130.
3. Zheng L, Wei H, He P et al (2016). Effects of Supplementation of Branched-Chain Amino Acids to Reduced-Protein Diet on Skeletal Muscle Protein Synthesis and Degradation in the Fed and Fasted States in a Piglet Model. Nutrients, 9(1): E17.
4. Contrusciere V, Paradisi S, Matteucci A et al (2010). Branched-chain amino acids in-duce neurotoxicity in rat cortical cultures. Neurotox Res, 17(4): 392-398.
5. Fernstrom JD (2005). Branched-chain ami-no acids and brain function. J Nutr, 135(6 Suppl): 1539S-1546S.
6. Kim J, Lee J, Kim S et al (2016). Exercise-induced rhabdomyolysis mechanisms and prevention: A literature review. J Sport Health Sci, 5(3): 324-333.
7. Sung DJ, Lee M, Park JK et al (2018). Com-bination of Antidepressant and Alcohol Intake as a Potential Risk Factor for Rhabdomyolysis. Iran J Public Health, 47(9): 1424-1425.
8. Holt, SG, Moore KP (2001). Pathogenesis and treatment of renal dysfunction in rhabdomyolysis. Intensive Care Med, 27(5): 803-811.
9. Zhenyukh O, Civantos E, Ruiz-Ortega M et al (2017). High concentration of branched-chain amino acids promotes oxidative stress, inflammation and migra-tion of human peripheral blood mono-nuclear cells via mTORC1 activation. Free Radical Biol Med, 104: 165-177.
10. Li T, Zhang Z, Kolwicz SC Jr et al (2017). Defective Branched-Chain Amino Acid Catabolism Disrupts Glucose Metabo-lism and Sensitizes the Heart to Ischemia-Reperfusion Injury. Cell Metab, 25(2): 374-385.
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| Issue | Vol 51 No 7 (2022) | |
| Section | Letter to the Editor | |
| DOI | https://doi.org/10.18502/ijph.v51i7.10104 | |
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How to Cite
1.
Jeon Y-K, Choi J, Sung DJ. Potential Influence of Exercise-Induced Rhabdomyolysis by Branched-Chain Amino Acid Supplementation. Iran J Public Health. 2022;51(7):1681-1682.
