Letter to the Editor

The p22phox Polymorphism C242T Is Not Associated with Peripheral Malondialdehyde and Superoxide Dismutase Levels in Healthy Korean Adults

Abstract

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1. Groemping Y, Rittinger K (2005). Activation and assembly of the NADPH oxidase: a structural perspective. Biochem J, 386(3):401-16.
2. Guzik TJ, Mussa S, Gastaldi D, et al (2002). Mechanisms of increased vascular super-oxide production in human diabetes mellitus: role of NAD(P)H oxidase and endothelial nitric oxide synthase. Circula-tion, 105(14):1656-62.
3. Rae J, Noack D, Heyworth PG, et al (2000). Molecular analysis of 9 new families with chronic granulomatous disease caused by mutations in CYBA, the gene encoding p22 (phox). Blood, 96(3):1106-12.
4. Guzik TJ, West NE, Black E, et al (2000). Functional effect of the C242T polymor-phism in the NAD (P)H oxidase p22phox gene on vascular superoxide production in atherosclerosis. Circulation, 102(15):1744-47.
5. Petrovič D (2014). Association of the -262C/T polymorphism in the catalase gene promoter and the C242T polymor-phism of the NADPH oxidase P22phox gene with essential arterial hypertension in patients with diabetes mellitus type 2. Clin Exp Hypertens, 36(1):36-9.
6. Li P, Qiu T, Qin C (2015). NADPH oxidase p22phox C242T polymorphism and is-chemic cerebrovascular disease: an updat-ed meta-analysis. Med Sci Monit, 21:231-8.
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IssueVol 48 No 5 (2019) QRcode
SectionLetter to the Editor
DOI https://doi.org/10.18502/ijph.v48i5.1832
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How to Cite
1.
CHO S-Y, SO W-Y, ROH H-T. The p22phox Polymorphism C242T Is Not Associated with Peripheral Malondialdehyde and Superoxide Dismutase Levels in Healthy Korean Adults. Iran J Public Health. 2019;48(5):977-979.